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Indeed, Sp was able to restore cytosolic and mitochondrial redox balance in neuronal cell line SHSY-5Y subjected to 1-methyl-4-phenylpyridinium (MPP + ) treatment which is known to cause mitochondria dysfunction and oxidative stress through complex I inhibition 1 (Fig

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doi: 10.1111/IJCP.12361 113 HeLWangJPingFYangNHuangJLiYet al

RPMI-1640 medium, high-glucose DMEM were obtained from Thermo Fisher Scientific Inc